Documentos de Académico
Documentos de Profesional
Documentos de Cultura
Synonyms
cardiogenic shock
cardiac shock
global hypo perfuse
cardiac gallop
Meaning
Cardiogenic shock is
characterized by a decreased
pumping ability of the heart that
causes a shock like state (i.e.,
global hypo perfusion). It most
commonly occurs in association
with, and as a direct result of,
acute myocardial infarction(AMI).
Incidence
Cardiogenic shock occurs in 8.6%
of patients with ST-segment
elevation
MI with 29% of those presenting to
the hospital already in shock.
It occurs only in 2% of nonSTsegment elevation MI.
Mortality/Morbidity
Leading cause of death in AMI.
The overall in-hospital mortality rate is
57%.
Persons older than 75 years, the
mortality rate is 64.1%.
Younger than 75 years, the mortality rate
is 39.5%.
Overall mortality when revascularization
occurs is 38%. If not attempted, mortality
rates approach 70%.
Race
Hispanics
- 74%
African Americans - 65%
Whites
-56%
Asians/others
- 41%.
Indians
- 43.7%
Sex
Women comprise 42% of all
cardiogenic shock patients.
Risk factors
Etiology
Acute myocardial ischemia
Pathophysiology
AMI
A systemic inflammatory
response syndrome
Myocardial infections
Myocardial ischemia
Other pathophysiological
mechanisms
papillary muscle rupture leading
to acute mitral regurgitation
(4.4%);
ventricular septal defect (1.5%)
wall rupture (4.1%) as a
consequence of AMI.
Medications
Clinical manifestations
The physical examination
findings are consistent with
shock.
Patients are in frank distress
profoundly diaphoretic with
mottled extremities
visibly dyspneic
Diagnostic measures
History collection
General physical examination
Initial vital sign assessment
Neck examination may reveal jugular
venous distention
LV dysfunction, characterized by
pulmonary edema, can be
auscultated as crackles with or
without wheezing.
Lab Studies
No one test is completely sensitive,
laboratory studies are directed at the
potential underlying cause.
Following are assessed in cases of
suspected cardiac ischemia:
Cardiac enzymes (eg, creatine kinase,
troponin, myoglobin)
CBC
Electrolytes
Coagulation profile (eg, prothrombin time,
activated partial thromboplastin time)
Imaging Studies
Portable chest radiograph
Overall impression of the cardiac
size
Pulmonary vascularity
Coexistent pulmonary pathology
A rough estimate of Mediastinal
and aortic sizes
Other Tests
ECG
Helpful if it reveals an acute injury
pattern consistent with an AMI
Echocardiogram
*To reveal akinetic or dyskinetic
areas of ventricular wall motion.
*To reveal surgically correctable
causes, such as valvular dysfunction
and tamponade.
Differential Diagnosis
MANAGEMENT
Prehospital Care: aimed at minimizing
any further ischemia and shock.
All patients require intravenous access, highflow oxygen administered by mask, and
cardiac monitoring.
Twelve-lead electrocardiography, The ED
physician, can thus be alerted, and may
mobilize the appropriate resources.
Pharmacotherapy
Aim: To reduce morbidity and to prevent
complications.
Intravenous vasopressors provide
inotropic support increasing perfusion
of the ischemic myocardium and all
body tissues.
Extreme heart rates should be avoided
because they may increase myocardial
oxygen consumption
2.Dobutamine, inamrinone
(formerly amrinone), or milrinone
may provide inotropic support. In
addition to their positive inotropic
effects, inamrinone and milrinone
have a beneficial vasodilator
effect, which reduces preload and
after load.
Dose: 5-20 mcg/kg/min IV
continuous infusion.
4.Natrecor (nesiritide)
Should be used with caution in the
setting of cardiogenic shock because
it has been shown to cause
hypotension.
5.Vasodilators
Smooth-muscle relaxants and
vasodilators that can reduce systemic
vascular resistance, allowing more
forward flow and improving cardiac
output.
Complications
Cardiopulmonary arrest
Dysrhythmia
Renal failure
Multisystem organ failure
Ventricular aneurysm
Thromboembolic sequelae
Stroke
Death
Prognosis
The prognosis is universally poor.
The mortality rate is more than
55% in patients treated medically.
At best, the rate is 38% in whom
surgical reperfusion is achieved.
NURSING MANAGEMENT
Assessment:
ABC,s,
tissue perfusion,
vital signs,
capillary refill ,
skin and urinary output.
Nursing Diagnosis
Decreased cardiac output related to shock
as manifested by increased diastolic BP,
tachycardia, dry mucous membrane,
pallor, cyanosis, cool and clammy skin.
Fear and anxiety related to severity of the
condition as manifested by verbalization of
anxiety about condition and fear of death
or withdrawal with no communication,
increase in heart and respiratory rate.
High risk for organ dysfunction related to
decreased tissue perfusion.
Nursing implementation
Health Promotion: Planning is
essential to help to prevent shock after
a susceptible individual has been
identified.
The primary goal for the patient with an
acute MI is to limit the size of the
infarction.
The nurse can modify the patients
environment to provide care at
intervals to increase patients oxygen
demand.
Acute Intervention:
The role of nurse in shock involves
monitoring the patients ongoing physical
and emotional status .
Planning and implementing the nursing
interventions and therapy
Evaluating the patients response to
therapy
Providing emotional support to patient and
family
Collaborating with other members of
health team.
THANK YOU