Está en la página 1de 7

Australian Dental Journal

The official journal of the Australian Dental Association


Australian Dental Journal 2017; 62:(1 Suppl): 51–57

doi: 10.1111/adj.12475

Retention and relapse in clinical practice


SJ Littlewood,*† S Kandasamy,‡§¶ G Huangk
*St Luke’s Hospital, Little Horton Lane, Bradford, BD5 0NA, United Kingdom.
†Leeds Dental Institute, University of Leeds, United Kingdom.
‡School of Dentistry, The University of Western Australia.
§Centre for Advanced Dental Education, Saint Louis University, MO, USA.
¶West Australian Orthodontics, Midland, WA, Australia.
kProfessor and Chair, University of Washington, Department of Orthodontics, Seattle, Washington, USA.

ABSTRACT
Maintaining teeth in their corrected positions following orthodontic treatment can be extremely challenging. Teeth have
a tendency to move back towards the original malocclusion as a result of periodontal, gingival, occlusal and growth
related factors. However, tooth movement can also occur as a result of normal age changes. Because orthodontics is
unable to predict which patients are at risk of relapse, those which will remain stable and the extent of relapse that will
occur in the long-term, clinicians need to treat all patients as if they have a high potential to relapse. To reduce this risk,
long term retention is advocated. This can be a significant commitment for patients, and so retention and the potential
for relapse must form a key part of the informed consent process prior to orthodontic treatment.It is vital that patients
are made fully aware of their responsibilities in committing to wear retainers as prescribed in order to reduce the chance
of relapse. If patients are unable or unwilling to comply as prescribed, they must be prepared to accept that there will be
tooth positional changes following treatment. There is currently insufficient high quality evidence regarding the best type
of retention or retention regimen, and so each clinician’s approach will be affected by their personal, clinical experience
and expertise, and guided by their patients’ expectations and circumstances.
Keywords: Fixed retainers, incisor crowding, relapse, removable retainers, retainers, retention, stability.
Abbreviations and acronyms: IPR = Interproximal reduction; SDB = Sleep disordered breathing.

will discuss the responsibilities of the patient and the


INTRODUCTION
clinician in reducing relapse following treatment.
Maintaining teeth in their corrected positions after
treatment is often the most challenging part of an
HISTORICAL PERSPECTIVE
orthodontic treatment plan. Relapse following
orthodontic treatment is traditionally thought of as a In 1934, Oppenheim stated “Retention is one of the
move back towards the original malocclusion. How- most difficult problems in orthodontia; in fact, it is
ever, a return towards the initial malocclusion does the problem.”1 Eighty years later, clinicians continue
not always occur, and relapse could be considered as to struggle with the same issue. Over the decades,
any unfavourable change in tooth position after many theories have been proposed regarding reten-
orthodontic treatment away from a corrected maloc- tion. For example, Kingsley felt that occlusion was
clusion. These changes may also be the result of nor- the key to stability.2 An alternative theory was that
mal age-related effects. the apical base had to be respected.3 Similarly, was
Clinicians involved in orthodontic treatment need the idea that the mandibular incisors had to be placed
to have a thorough understanding of the aetiology of over basal bone in order to promote stability.4–6
relapse and be familiar with different methods of Finally, Rogers proposed that proper function and
reducing this relapse. This includes familiarity with balance of the musculature was related to stability.7
the advantages and disadvantages of various retainers, These theories, as well as others, will be discussed in
as well as the ability to advise patients on how to more detail below. In reality, orthodontic relapse is
wear retainers effectively. complicated and highly variable, and better data to
The present article will provide a contemporary provide evidence-based recommendations to our
overview of retention and relapse in orthodontics, and patients, is needed.
© 2017 Australian Dental Association 51
SJ Littlewood et al.

The best information currently available comes activity of a healthy periodontium will resist proclina-
from the long-term post-retention registry at the tion of the teeth.15 The farther teeth are moved out of
University of Washington. Riedel and Little are cred- this ‘neutral’ zone, the more unstable they are likely
ited with the collection of over 800 long-term post- to be. This is particularly true for the lower labial seg-
retention cases and discovered that relapse occurred ment and if incisors are proclined or retroclined exces-
in a high percentage of patients but in an individual sively, relapse is more likely. It is also believed that
patient, relapse was quite unpredictable.8,9 It was also significant changes in the arch form, in particular the
found that canine width expansion was unstable in lower intercanine width, make relapse due to soft tis-
the long-term9, and Little stated that lifetime perma- sue pressures more likely. There are, of course, situa-
nent retention was the only reliable way to prevent tions in which it is necessary to alter lower incisor
relapse.10 position or intercanine width, for example, to improve
aesthetics, but, in these cases, the clinician needs to
plan an appropriate retention strategy to resist the
AETIOLOGY OF RELAPSE AND DURATION OF
increased relapse potential.16–18
RETENTION
Relapse after orthodontic treatment can be a result of
Physiological relapse
orthodontic factors and normal age changes.11,12
These orthodontic factors include periodontal and gin- It has been shown that there are potential subtle facial
gival factors, occlusal factors and factors related to growth or age-related changes that occur throughout
soft tissue pressures and limits of the dentition. life19 including minor changes in the relationship
between the mandible and maxilla, and changes in the
soft tissue pressures on the dentition. The dentition is
Periodontal and gingival factors
therefore within a biological environment that is con-
When teeth are moved the tissues in the periodontal stantly changing, and so it is not surprising that there
ligament and gingivae remodel to the new tooth posi- is the potential for changes to occur in the alignment
tion. Until these tissues have remodelled, they have a of teeth and occlusal relationships throughout life.
tendency to manoeuvre the teeth back towards their These changes in alignment and occlusal relationships
original position. The fibres that take the longest to should perhaps be regarded as normal age changes.
remodel are the elastic fibres around the neck of the
teeth, the dento-gingival and interdental fibres, which
INFORMED CONSENT
can take 8 months or more to remodel.13 The teeth
therefore need to held in position for long enough for Whilst relapse does not happen in every patient, clini-
these fibres to adjust. An alternative approach is to cally it is difficult to predict which patients will
use a simple surgical procedure called pericision that undergo post-treatment change and so it is critical
severs these gingival fibres and this will be discussed that all patients are treated as if they have the poten-
later.14 tial for relapse. As a result, many clinicians now rec-
ommend life-long retention. When a patient asks
“How long should I wear my retainers”, the answer
Occlusal factors
is “For as long as you want straight teeth”. An impor-
It is purported that a soundly interdigitated dentition, tant aspect of informed consent for orthodontic treat-
with even occlusal contacts and correct occlusal load- ment is the need for the patient to fully understand
ing of teeth, is more likely to be stable; however, there the long-term risk of relapse, and appreciate the pro-
is no substantial agreement or evidence to support this cedures to minimise the risk. There are important
claim. It must be recognised that gross occlusal inter- responsibilities for the clinician and the patient.
ferences, displacing tooth contacts and the abnormal The clinician’s responsibility is to explain the unpre-
loading of teeth may predispose the affected teeth to dictable nature of relapse, the factors known to be
mobility which may contribute to relapse. involved and advise on the reduction of risk by the
appropriate use of retainers. The clinician needs to
explain the commitment that is required, including
Soft tissue pressures and limits of the dentition
any possible long-term financial costs associated with
It is preferable, where possible, to position teeth repairing and replacing retainers, costs associated with
within an area of soft tissue balance between the ton- addressing any relapse that occurs following failure to
gue on the lingual and the lips and cheeks on the comply with retention, and provide information about
labial aspect. This is an area of balance that is retainer care. The long-term care of retainers is vital
prosthodontically referred to as the neutral zone. to ensure that they fulfil their maintenance role in the
Although the forces from the tongue are stronger, the long-term, without compromising oral health. This
52 © 2017 Australian Dental Association
Relapse and retention in orthodontics

advice should include a retainer review program to (a)


assess the need for repair or replacement.
It is also important that patients understand their
responsibility and involvement in reducing relapse.
Before the commencement of treatment, patients
should be fully informed about the retainer commit-
ment required.

TYPES OF RETAINERS

Removable Retainers
Removable retainers have the advantages of being
easier to maintain oral hygiene (as they can be
removed for cleaning), and may only need to be worn
part-time. It has been shown that in many cases,
removable retainers need only be worn at night to
maintain dental stability.20–22 Good patient compli-
ance is essential with removable retainers, and if con- (b)
sistent wear is overlooked, relapse occurs. This
method of retention places full responsibility directly
on the patient in maintaining tooth alignment follow-
ing orthodontic treatment.
The most common examples of removable retainers
used worldwide are the Hawley-type retainers (with
an acrylic baseplate and usually a wire labial bow,
Figure 1) and thermoplastic retainers (made from
clear plastic, Figure 2). There is some evidence to sug-
gest that, at least in the short-term, patients prefer the
appearance and comfort of thermoplastic retainers
which are more cost-effective and slightly more effec-
tive in maintaining stability, particularly in the lower
Fig. 1 (a) Upper Hawley style removable retainer. (b) Lower Hawley
arch.23,24 There is no high quality, long-term research style removable retainer.
to indicate whether these advantages are maintained
long-term.

Fixed retainers
Fixed retainers offer the advantage of being in place
permanently which removes the need for patient com-
pliance with retainer wear (Figure 3, the retainers are
typically bonded to the palatal/lingual surfaces of the
labial segments. As they cannot be removed for clean-
ing, they are more prone to plaque and calculus accu-
mulation.25 It is therefore vital that patients are
provided with clear instructions on oral hygiene mea-
sures associated with their bonded retainers. The
retainers also need to be checked regularly to ensure
that they are still bonded in place. In addition, there
are reports of occasional, severe, unwanted tooth
movements caused by different types of failed/faulty Fig. 2 Upper and lower clear thermoplastic removable retainers.
fixed retainers as a result of the bonding of some or
all teeth within the span of the fixed retainer.26–28
This method of retention makes the clinician responsi- are made aware that if appliance maintenance is not
ble for the maintenance of the fixed retainer. It is crit- performed by either their orthodontist or general den-
ical during the informed consent process that patients tist, they are at risk of tooth relapse.
© 2017 Australian Dental Association 53
SJ Littlewood et al.

(a) high-quality evidence on the best type of retainer or


retention regimen.29 As a result, each clinician’s
approach to retention will be strongly influenced by
their clinical experience and expertise with different
retainers, as well as the patient’s values, expectations
and circumstances.

ADJUNCTIVE PROCEDURES
Adjunctive procedures are techniques that alter the
hard or soft tissues in an attempt to reduce relapse.
(b) Examples include pericision and interproximal reduc-
tion.

Pericision
Pericision is a simple soft tissue surgical technique
aimed at severing the periodontal fibres around the
neck of the teeth (dento-gingival and interdental
fibres) and is sometimes referred to as supracrestal cir-
cumferential fiberotomy. The procedure is performed
under local anaesthetic and there is weak evidence to
Fig. 3 (a) Lower fixed retainer; Bonded only at teeth 33 and 43.
(b) Lower fixed retainer; Bonded at all the lower incisor teeth.
suggest that it reduces relapse of rotated teeth, partic-
ularly in the maxilla.14,30 The procedure should only
be undertaken in cases in which there is a good gingi-
Studies which have compared the merits of fixed val biotype and cortical bone support, minimal or no
and removable retainers suggest that both appear to recession and the patient has excellent oral hygiene.
reduce relapse in routine cases. However, there are As it is a surgical procedure, it is usually reserved for
situations in which full-time retention is required, and severely rotated teeth.
so a fixed retainer may be preferred. The main exam-
ples of higher-risk relapse cases are listed in Table 1.
Interproximal reduction
Clinicians may often choose to use a combination
of fixed and removable retainers in a process referred Interproximal reduction (IPR) is a hard tissue proce-
to as ‘dual’ retention. The patient is fitted with a fixed dure aimed at removing small amounts of enamel
retainer, and is provided with a removable retainer to interdentally. It is not fully understood how this may
wear at night as a back-up. reduce relapse, but it has been suggested that it may
A recent Cochrane Review which assessed the meth- flatten the interdental contacts between incisors and
ods of retention reported that there was insufficient therefore increase stability. It is suggested that IPR
may compensate for the normally occurring reduction
in inter-canine width which occurs during adoles-
Table 1. Cases where fixed retention may be cence.31
preferred A recent randomised controlled trial compared the
use of interproximal reduction with other methods of
• Following closure of a spaced dentition (including a marked retention for Class I crowded cases treated with
median diastema) extractions and fixed appliances.32–34 It was shown
• Following creation of space prior to prosthodontic
that using IPR alone in the lower arch (without a
management
• Reduced periodontal support retainer) was equally successful at reducing relapse as
• Following correction of severe rotations using a lower bonded retainer or a positioner. This
• For patients who cannot tolerate even minor changes in
raises the interesting possibility of the use of inter-
occlusion
• Following correction of severely impacted teeth proximal reduction as an alternative or in conjunction
• In cleft lip and palate patients with evidence of severe post- with retainers. The results need to be interpreted with
surgical scarring that may predispose to relapse after
orthodontics
caution before deciding on universally applying this
• In compromised cases in which the aims of treatment may be procedure on all patients. Consideration needs to be
more limited, and focus is geared more towards achieving a given to the data which refers to patients who were
good aesthetic result without aiming for a fully corrected
malocclusion with ideal occlusion
followed for 5 years as it is possible that relapse
might occur after this observation period.
54 © 2017 Australian Dental Association
Relapse and retention in orthodontics

transverse discrepancy between the maxillary and


OTHER CONSIDERATIONS AFFECTING RELAPSE
mandibular arches. There is a current trend towards
treatment planning around expanding the maxillary
Third molars
arch (even when there is no transverse discrepancy)
The extraction of mandibular third molars to prevent with the aim of treating malocclusions ‘non-extrac-
late lower incisor crowding remains a controversial tion’ or, at least, without premolar teeth extrac-
topic and a common practice in dentistry. From an tions.50 As any expansion (especially in the
orthodontic perspective, third molars have essentially intercanine region) of the mandibular arch is essen-
little to do with lower incisor crowding. Multiple tially unstable, the mandibular arch is considered the
studies have investigated the influence of third molars best guide for gauging the success of expansion.
on crowding and assessed their effects on the lower Expansion of the maxillary arch, especially when
dental midline, anterior crowding and cases of bilat- there is no buccal crossbite present will require the
eral and unilateral third molar agenesis.35–39 The mandibular arch to be expanded accordingly, which is
results found only a small (negligible clinical signifi- inherently an unstable procedure.51
cance) or no effect with large standard deviations. Although there is a lack of strong evidence regard-
The large standard deviations indicated that, in some ing the long-term stability of maxillary expansion,
cases, the crowding was greater in patients without one must be mindful of the limits the dentition when
third molars present. Late incisor crowding is multi- carrying out expansion in either arch.52–54 Expansion
factorial in nature and factors other than third molars carried out in the maxillary arch should only result in
play an important role. The removal of third molars minimal expansion in the lower arch to provide the
on the sole basis of preventing lower incisor crowding patient with the best chance of long term stability.
is unsubstantiated and not evidence based.40
Expansion and Sleep Disordered Breathing
Growth considerations
A narrow maxilla has been suggested as a predispos-
There is no doubt that any skeletal changes that occur ing craniofacial morphological risk factor related to
following orhodontic treatment will influence the sleep disordered breathing (SDB). While studies have
achieved dental relationships. The patient’s growth shown improvement in airway dimensions and a
pattern is usually taken into consideration before and reduction in nasal resistance following maxillary
during treatment; however, little consideration is usu- expansion55–57 most have shown some relapse to pre-
ally given to post treatment skeletal growth changes expansion values at review.58–60 Many of the studies
on the final outcome.41–45 Important skeletal changes unfortunately have methodological flaws, primarily
that require consideration and patient education are related to the lack of long-term follow up, the use of
extremes in vertical facial patterns, deep bites and small heterogenous samples without a control group,
anterior open bites, late growth in long-face and Class differences in the amount of expansion in each subject
III patients and skeletal changes related to post- and the large individual variability observed which
orthognathic surgical patients including condylar has over-exaggerated the effects of expansion on the
resorption. airway.
Recent studies have shown a significant improve-
ment in apnoea-hyponoea index scores in children
Maxillary and mandibular expansion
with sleep apnoea following rapid maxillary expan-
Longitudinal studies about dental arch dimensions in sion.61,62 As a result, rapid maxillary expansion was
untreated subjects have shown that there is an advocated before adenotonsillectomy in children as
increase in intercanine and intermolar widths until the there was the prospect of a reduced need for surgery
complete eruption of the permanent dentition follow- following expansion in a large proportion of cases.
ing which a decrease in dental widths, more in the According to the inclusion criteria, it appeared that
intercanine than intermolar widths, occurs. The width maxillary expansion may have been carried out with-
reduction continues for many decades even up to the out the presence of a posterior crossbite and based on
eighth decade of life.46–49 Therefore, any orthodontic the presence of a high palatal vault; narrow maxillary
dental expansion beyond the original pre-treatment arch related to the restriction of the upper jaw at its
status will increase the relapse potential post-treat- base.61,62 Clearly, advocating maxillary expansion pri-
ment and hence justify the need for life-time reten- marily to address airway issues or SDB when there is
tion. no obvious posterior crossbite, is not evidence based
Skeletal maxillary expansion is generally carried out or justified as this would lead to significant bite open-
as part of an orthodontic treatment plan to primarily ing, posterior scissor bites and unfavourable den-
address an insufficient maxillary width and resulting toalveolar compensations.
© 2017 Australian Dental Association 55
SJ Littlewood et al.

11. Littlewood SJ, Russell JS, Spencer RS. Why do orthodontic


CONCLUSIONS cases relapse? Orthodontic Update 2009;2:43–49.
Maintaining teeth in their corrected positions follow- 12. Melrose C, Millet DT. Toward a perspective on orthodontic
retention? Am J Orthod Dentofac Orthop 1998;113:507–514.
ing orthodontic treatment can be extremely challeng-
13. Reitan K. Clinical and histologic observations on tooth move-
ing. Relapse after orthodontic treatment is the result ment during and after orthodontic treatment. Am J Orthod
of teeth moving back towards the original malocclu- 1967;53:721–745.
sion, but changes in tooth position may also occur as 14. Edwards JG. A long term prospective evaluation of the circum-
a normal part of the growth and aging process. ferential supracrestal fiberotomy in alleviating orthodontic
relapse. Am J Orthod Dentofac Orthop 1988;93:380–387.
Relapse is also unpredictable, and so it should be pre-
15. Proffit WR. Equilibrium theory revisited: factors influencing
sumed that every patient has the potential for long- position of the teeth. Angle Orthod 1978;48:175–186.
term changes. As part of the informed consent process 16. De La Cruz A, Little RM, Sampson P,  Artun J, Shapiro PA.
for orthodontic treatment, patients need to be fully Long-term changes in arch form after orthodontic treatment and
aware of their commitment to wear retainers for as retention. Am J Orthod Dentofac Orthop 1995;107:518–530.
long as they want to keep their teeth in their corrected 17. Burke SP, Silveira AM, Goldsmith LJ, Yancey JM, Stewart AV,
positions. It is the clinician’s responsibility to ensure Scarfe WC. A meta-analysis of mandibular intercanine width in
treatment and postretention. Angle Orthod 1997;68:53–60.
that patients are appropriately instructed regarding
18. Little RM. Stability and relapse of dental arch alignment. Br J
the care of their retainers and provided advice about Orthod 1990;17:235–241.
the timing of retainer review and by whom. When 19. Behrents RG, Harris EF, Vaden JL, Williams RA, Kemp DH.
using fixed retainers, as part of the treatment consent Relapse of orthodontic treatment results: growth as an etiologi-
process, patients must be informed of the importance cal factor. J Charles H Tweed Int Found 1989;17:65–80.
of maintaining the integrity of the fixed retainer and 20. Gill DS, Naini FB, Jones A, Tredwin CJ. Part-time versus full-
time retainer wear following fixed appliance therapy: a random-
who will be taking this long-term responsibility. There ized prospective controlled trial. World Journal of Orthodontics
is currently insufficient high quality evidence on the 2007;8:300–306.
best type of retention or retention regimen, so each 21. Shawesh M, Bhatti B, Usmani T, Mandall N. Hawley retainers
clinician’s approach to retention will be affected by full or part time? A randomized clinical trial. Eur J Orthod
2010;32:165–170.
personal clinical experience and expertise with differ-
22. Thickett E, Power S. A randomized clinical trial of thermoplas-
ent retainers, and also the patients’ expectations and tic retainer wear. Eur J Orthod 2010;32:1–5.
circumstances. Patients who are unable or unwilling
23. Hichens L, Rowland H, Williams A, et al. Cost-effectiveness
to wear their retainers as prescribed must be prepared and patient satisfaction: Hawley and vacuum-formed retainers.
to accept that there will be relapse following Eur J Orthod 2007;29:372–8.
orthodontic treatment. The extent of the relapse is 24. Rowland H, Hichens L, Williams A, et al. The effectiveness of
unpredictable. Hawley and vacuum- formed retainers: A single-center random-
ized controlled trial. Am J Orthod Dentofac Orthop
2007;132:730–7.
25. Millett DT, McDermott P, Field D, et al. Dental and Periodon-
REFERENCES tal Health with Bonded or Vacuum-formed Retainer. In: IADR
1. Oppenheim A. The crisis in orthodontia. Part I. Tissue changes Conference Abstract 3168. Toronto, 2008
during retention. Int J Orthod 1934;6:639–644. 26. Abudiak H, Shelton A, Spencer RJ, Burns L, Littlewood SJ. A
2. Kingsley N. A Treatise on Oral Deformities as a Branch of complication with orthodontic fixed retainers: A case report.
Mechanical Surgery. New York: Appleton & Co, 1880. Ortho Update 2011;4:112–117.
3. Lundstr€
om A. Malocclusions of the teeth regarded as a problem 27. Kucera J, Marek I. Unexpected complications associated with
in connection with the apical base. Int J Orthod Oral Surg mandibular fixed retainers: A retrospective study. Am J Orthod
1925;11:591. Dentofacial Orthop 2016;149:202–11.
4. Grieve GW. The stability of the treated denture. Am J Ortho 28. Shaughnessy TG, Proffit WR, Samara SA. Inadvertent tooth
Oral Surg 1944;30:171–195. movement with fixed lingual retainers. Am J Orthod Dentofa-
cial Orthop 2016;149:277–86.
5. Tweed CH. Why I extract teeth in the treatment of certain
types of malocclusion. Alpha Omegan 1952;46:93–104. 25. 29. Littlewood SJ, Millett DT, Doubleday B, Bearn DR, Worthing-
ton HV. Retention procedures for stabilising tooth position
6. Tweed CH. Indications for extraction of teeth in orthodontic after treatment with orthodontic braces. Cochrane Database of
procedure. Am J Orthod Oral Surg 1944;30:405–428. Systematic Reviews 2016, Issue 1. Art. No.: CD002283. DOI:
7. Rogers AP. Making facial muscles our allies in treatment and 10.1002/14651858.CD002283.pub4.
retention. Dent Cosmos 1922;64:711–730. 30. Redlich M, Rahamim E, Gaft A, Shoshan S. The response of
8. Little RM, Wallen TR, Riedel RA. Stability and relapse of supraalveolar gingival collagen to orthodontic rotation move-
mandibular anterior alignment- first premolar extraction cases ment in dogs. Am J Orthod Dentofac Orthop 1996;110:247–255.
treated by traditional edgewise orthodontics. Am J Orthod 31. Aasen TO, Espeland L. An approach to maintain orthodontic
1981;80:349–365. alignment of lower incisors without the use of retainers. Eur J
9. Little RM, Riedel RA, Artun J. An evaluation of changes in Orthod 2005;27:209–214.
mandibular anterior alignment from 10 to 20 years postreten- 32. Edman Tynelius G, Bondemark L, Lilja-Karlander E. Evalua-
tion. Am J Orthod Dentofacial Orthop 1988;93:423–428. tion of orthodontic retention capacity after one year of reten-
10. Little RM. Clinical implications of the University of Washing- tion - a randomized controlled trial. Eur J Orthod 2010; 32:
ton post-retention studies. J Clin Orthod 2009;43:645–51. 542–7.

56 © 2017 Australian Dental Association


Relapse and retention in orthodontics

33. Edman Tynelius G, Bondemark L. A randomized controlled 51. Gianelly A. Evidence-based therapy: an orthodontic dilemma.
trial of three orthodontic retention methods in Class I four pre- Am J Orthod Dentofacial Orthop 2006;129:596–8.
molar extraction cases - stability after 2 years in retention. 52. Lagravere MO, Heo G, Major PW, Flores-Mir C. Meta-analysis
Orthod Craniofac Res 2013; 16: 105–115. of immediate changes with rapid maxillary expansion treat-
34. Edman Tynelius G, Petren S, Bondemark L, Lilja-Karlander ment. J Am Dent Assoc 2006;137:44–53.
E. Five-year postretention outcomes of three retention meth- 53. Lagravere MO, Major PW, Flores-Mir C. Long-term dental
ods—a randomized controlled trial. Eur J Orthod 2015; 37: arch changes after rapid maxillary expansion treatment: a sys-
345–353. tematic review. Angle Orthod 2005;155–61.
35. Ades AA, Joondeph DR, Little RM, Chapko MK. A long-term 54. Marshall SD, Shroff B. Long-term skeletal changes with rapid
study of the relationship of third molars to the changes in the maxillary expansion: a review of the literature. Semin Orthod
mandibular dental arch. Am J Orthod Dentofac Orthop 2012;18:128–133.
1990;97:323–335.
55. Cordasco G, Nucera R, Fastuca R, et al. Effects of orthopedic
36. Harradine NWT, Pearson MH, Toth B. The effect of extraction maxillary expansion on nasal cavity size in growing subjects: A
of third molars on late lower incisor crowding: A randomized low dose computer tomography clinical trial. Int J Pediatr
controlled trial. Br J Orthod 1998;25:117–122. Otorhinolaryngol 2012;76:1547–1551.
37. Kaplan RG. Mandibular third molars and postretention crowd- 56. Monini S, Malagola C, Villa MP, et al. Rapid maxillary expan-
ing. Am J Orthod Dentofac Orthop 1974;66:411–430. sion for the treatment of nasal obstruction in children younger
38. Lindquist B, Thilander B. Extraction of third molars in cases of than 12 years. Arch Otolaryngol Head Neck Surg
anticipated crowding in the lower. Am J Orthod Dentofac 2009;135:22–7.
Orthop 1982;81:130–139. 57. Baratieri C, Alves M Jr, de Souza MMG, de Souza Ara
ujo MT,
39. Richardson M. Lower molar crowding in the early permanent Maia LC. Does rapid maxillary expansion have long-term
dentition. Angle Orthod 1985;55:51–57. effects on airway dimensions and breathing? Am J Orthod
40. Kandasamy S. Counterpoint: Asymptomatic third molars: eval- Dentofacial Orthop 2011;140:146–156.
uation and management. Am J Orthod Dentofac Orthop 58. Langer MRE, Itikawa CE, Valera FCP, Matsumoto MAN,
2011;140:11–17. Anselmo-Lima WT. Does rapid maxillary expansion increase
41. Nanda RS, Nanda SK. Considerations of dentofacial growth in nasopharyngeal space and improve nasal airway resistance? Int
long-term retention and stability: Is active retention needed? J Pediatr Otorhinolaryngol 2011;75:122–125.
Am J Orthod Dentofac Orthop 1992;101:297–302. 59. Gordon JM, Rosenblatt M, Witmans M, et al. Rapid Palatal
42. Nanda SK. Growth patterns in subjects with long and short Expansion Effects on Nasal Airway Dimensions as Measured
faces. Am J Orthod Dentofac Orthop 1990;98:247–258. by Acoustic Rhinometry. Angle Orthod 2009;79:1000–1007.
43. Nanda SK. Patterns of vertical growth in the face. Am J Orthod 60. Hartgerink DV, Vig PS, Orth D, Abbott DW. The effect of
Dentofac Orthop 1988;93:103–116. rapid maxillary expansion on nasal airway resistance. Am J
Orthod Dentofacial Orthop 1987;92:381–389.
44. Bjork A, Skieller V. Facial development and tooth eruption. An
implant study at the age of puberty. Am J Orthod Dentofac 61. Villa MP, Malagola C, Pagani J, et al. Rapid maxillary expan-
Orthop 1972; 62: 339–383. sion in children with obstructive sleep apnea syndrome: 12-
month follow-up. Sleep Med 2007;8:128–134.
45. Bjork A, Skieller V. Normal and abnormal growth of the mand-
ible. A synthesis of longitudinal cephalometric implant studies 62. Pirelli P, Saponara M, Guilleminault C. Rapid maxillary expan-
over a period of 25 years. Eur J Orthod 1983;5:1–46. sion before and after adenotonsillectomy in children with
obstructive sleep apnea. Somnologie 2012;16:125–132.
46. Sillman JH. Dimensional changes of the dental arches: longitu-
dinal study from birth to 25 years. Am J Orthod Dentofac
Orthop 1964;50:824–842. Address for correspondence:
47. Bishara SE, Jakobsen JR, Treder J, Nowak A. Arch width Clinical Associate Professor Sanjivan Kandasamy
changes from 6 weeks to 45 years of age. Am J Orthod Dento- Department of Orthodontics
fac Orthop 1997;111:401–409.
School of Dentistry
48. Bishara SE, Jakobsen JR, Treder J, Nowak A. Arch length The University of Western Australia
changes from 6 weeks to 45 years. Am J Orthod Dentofac
Orthop 1998;68:69–74. 17, Monash Ave
49. Carter GA, McNamara JA Jr. Longitudinal dental arch changes Nedlands 6009
in adults. Am J Orthod Dentofac Orthop 1998;114:88–99. Western Australia
50. Rinchuse DJ, Kandasamy S. Implications of the inclination of Australia
the mandibular first molars in the “extractionist” versus “ex- Email: sanj@kandasamy.com.au
pansionist” debate. World J Orthod 2008;9:383–390.

© 2017 Australian Dental Association 57

También podría gustarte